Education 论文代写:Blood Lactate Heart Rate Physical
The subjects exercised until exhaustion on a cycle ergometer at a 60% VO2peak in a thermo neutral environment. IOP was measured before and after exercise and during recovery (15, 30 and 45 min) using the applanation tonometry method. Skin and rectal temperatures, heart rate and oxygen uptake were measured continuously. IOP was similar for the right eye and the left eye and increased post water ingestion under both exercising and resting conditions (P<0.05) but did not differ between resting and exercising situations, or between the three water temperatures. Time to exhaustion was not affected by the different water temperatures. Rectal temperature, hydration status, heart rate, oxygen uptake, carbon dioxide extraction and lactate concentration were increased by exercise but were not affected by water temperature. We conclude that IOP was not affected by exercise and that water ingestion increased IOP as expected, regardless of water temperature.
Wasserman, Connolly and Pagliassotti reports on the regulation of hepatic lactate balance during exercise. The rate of change of lactate across the liver gives important insights in to intracellular processes during muscular work. At the on set of exercise, hepatic glycogenolysis increase rapidly, resulting in high rates of glycolytic flux and a transient rise in lactate output. With increasing exercise duration, gluconeogenisis is accelerated and the liver gradually shifts from a lactate – producing to a lactate – consuming state. Exercise induced changes in hormone levels are critical in the regulation of hepatic glycogenolysis and guluconeogenesis and therefore, net hepatic lactate balance.
Hurley et al (1984) studied effects of blood lactate levels during sub maximal exercise. Eight men were studied before and after a 12-wk exercise program to determine the effect of training on blood lactate levels during sub maximal exercise. The training elicited a 26% increase in maximum O2 uptake (VO2max). Lactate concentrations at the same relative exercise intensities in the 55-75% of VO2max range were significantly lower after training. A significantly higher relative exercise intensity was needed to elicit a given lactate level in the 1.5- to 3.0-mM range after training. O2 uptake at the work rate required to raise blood lactate to 2.5 mM was 39% higher after training. A blood lactate of 2.5 mM was attained at 68 +/- 4% VO2max before and 75 +/- 3% of VO2max after training. Eight competitive runners required an even higher relative work rate (83 +/- 2% of VO2max) to attain a blood lactate of 2.5 mM.
Education 论文代写:Blood Lactate Heart Rate Physical
主体行使在热中性环境60% VO2peak在脚踏车疲惫。眼压测量运动前后及恢复期（15、30、45 min）使用压平眼压计法。皮肤和直肠温度，心脏率和氧的吸收量连续测量。眼压是相似的右眼和左眼和增加后水摄入的运动和休息条件（P＜0.05），但没有显着差异休息和运动的情况下，或之间的三个水的温度。时间不受不同的水温度的影响。运动，直肠温度，水化状态，心脏率，氧吸收，二氧化碳提取和乳酸浓度增加，但不受水温度。我们的结论是眼压没有受到影响，运动和水摄入增加眼压预期，无论水的温度。
沃瑟曼、康纳利和Pagliassotti的报告对肝乳酸平衡调节运动时。在肝脏中的乳酸的变化率提供了重要的见解，在肌肉工作过程中的细胞内过程。在开始锻炼，肝糖原分解迅速增加，导致高的糖酵解通量率和短暂的上升，乳酸产量。随着锻炼的持续时间，加速肝脏gluconeogenisis逐渐从乳酸生产乳酸消费状况。运动性激素水平变化的关键guluconeogenesis肝糖原分解和调控，因此，净肝乳酸平衡。
赫尔利等（1984）研究了亚最大运动过程中血乳酸水平的影响。八名男子进行了研究之前和之后的一个12周的运动计划，以确定训练对血乳酸水平在次最大运动。训练引起的最大氧气摄取增加26%（最大摄氧量）。在相同的相对运动强度在最大摄氧量范围55-75%乳酸浓度显著低于训练后。较高的相对运动强度需要引出一个给定的乳酸水平在1.5 -训练后切口范围。2吸收的工作速率需要提高血乳酸到2.5毫米是39%，较高的训练后。血乳酸2.5毫米达到68 + / – 4%最大摄氧量在75 + / – 3% VO2max训练后。八选手竞争的要求更高的相对速度（83 + / – 2%最大摄氧量）达到2.5毫米的血乳酸。